In this interview, return guest Stephanie Seneff, Ph.D., a senior research scientist at MIT for over five decades, discusses her paper, “Innate Immune Suppression by SARS-CoV-2 mRNA Vaccinations: The Role of G-quadruplexes, Exosomes and MicroRNAs,” …
In this interview, return guest Stephanie Seneff, Ph.D., a senior research scientist at MIT for over five decades, discusses her paper, “Innate Immune Suppression by SARS-CoV-2 mRNA Vaccinations: The Role of G-quadruplexes, Exosomes and MicroRNAs,” published in the June 2022 issue of Food and Chemical Toxicology.
In “Innate Immune Suppression by SARS-CoV-2 mRNA Vaccinations: The Role of G-quadruplexes, Exosomes and MicroRNAs,” Stephanie Seneff, Ph.D., and Drs. Peter McCullough, Greg Nigh and Anthony Kyriakopoulos explain how the COVID shots suppress your innate immune function, and how they may cause neurological diseases.
Their landmark paper was the source of major controversy in that the prominent journal in which it was published receive much negative feedback and the editor of the journal was forced to resign although the paper has not been retracted at this time.
G4s are genome-wide targets of transcriptional regulation. The “G” stands for guanine. G4 is DNA sequence of four consecutive guanines, which plays an important role in diseases such as cancers and neurological disorders. The COVID jab spike protein produces far more G-quadruplexes (G4) than the virus. The G4 causes prion protein to misfold, which can result in prion diseases such as Creutzfeldt-Jakob disease and Alzheimer’s.
Two specific microRNA have been found in people who got the jab, and these microRNA’s interfere with Type 1 interferon response, which is a key part of your immune system. When Type 1 interferon is suppressed, you become more prone to infection and chronic disease.
The COVID jab produces high levels of immunoglobulin (IgG) antibodies, which are associated with autoimmune disease. It does not produce mucosal antibodies.
Antibodies against the spike protein may be responsible for cases in which patients developed highly aggressive prion disease after their second jab.
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