Which Cells Do mRNA Vaccines Target? Dr Stanley Laham on mRNA autoimmune dangers
Which Cells Are Targeted? Dr Stanley Laham Ivermectin mRNA autoimmune dangers
University of Florida
There is also a predictable nefarious effect, that is the onset of an autoimmune cascade of events.
The mRNA will be translated in the cytoplasm of Cells it penetrates into viral proteins that can prime our immune system to make specific circulating antibodies to prevent the pathogenesis of the virus if exposed.
But this mRNA bray also be incorporated In our DNA by reverse transcription and remain as part of our genome. This would heathy Cells to make viral protein that migrate to the cell membrane. In this scenario, these cells would be recognized as foreign by our immune system and be attacked causing a new autoimmune disease. There is no way to predict over the months and years after vaccination with this viral mRNA how many cells in what organs would be endangered by making this foreign protein.
2-And this is a most important point: the formation of RNA-antigen complexes that is found in those peritoneal exudates macrophages as a precursor to the completed I-RNA strands was not inhibited by either Actinomycin D or rifampin suggesting that the RNA formed in those complexes during the antigen processing phase was not transcribed but formed in another manner, perhaps by Reverse Translatase.
3-The ability of I-RNA to stimulate antibody production in spleen cell cultures is not inhibited by Actinomycin D nor rifampicin indicating clearly that there is no need for transcription of any gene for the antibody produced. The information for the whole antibody must be completely contained in the I-RNA..
So the evidence suggests that some antigens are first sequestered by a specialized sub-population of macrophages that process them by first forming RNA-antigen complexes. That RNA does not need to be transcribed from any gene region since its formation is not blocked by inhibitors of RNA polymerase, but is perhaps elaborated by a Reverse Translatase that probably makes up the major protein component of those complexes. The next step seems to be incorporation of that RNA into a gene region by reverse transcriptase and then transcription of the complete strand of I-RNA. This I-RNA is released and picked up by B lymphocytes that can then initiate an antibody response. Once this I-RNA is incorporated in the appropriate gene region of the B cells by reverse transcription, they become committed memory cells capable of a more rapid secondary immune response if stimulated again by the antigen.
Proof of the existence of such an enzyme would be in RNA-DNA hybridization studies. If one could extract the RNA from the RNA-protein complexes that seem crucial as a precursor to the formation of I-RNA in those specialized cells and show that it would only hybridize with DNA from V or D gene segments of committed memory B cells but not unsensitized B cells from the same donor, it would be a powerful argument for the existence of reverse translation.
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