COVID Vaccine Shedding Tied To RSV And Polio Like Illnesses In Children

https://childrenshealthdefense.org/defender/chimpanzees-children-origins-respiratory-syncytial-virus/ (being blocked)
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1- "From Chimpanzees to Children: The Origins of RSV — Respiratory Syncytial Virus"
https://namelyliberty.com/from-chimpanzees-to-children-the-origins-of-rsv-respiratory-syncytial-virus/
2- Respiratory syncytial virus infection in children
https://pubmed.ncbi.nlm.nih.gov/21243988/
3- Why are child cases of RSV surging in the wake of COVID?
https://medicalxpress.com/news/2021-07-child-cases-rsv-surging-covid.html
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"COVID Vaccine Shedding Tied To RSV And Polio Like Illnesses In Children"
https://www.bitchute.com/video/WbmkRQWPfYVg/

From Chimpanzees to Children: The Origins of RSV — Respiratory Syncytial Virus
Source: Children's Health Defense
August 27, 2021 at 11:00 am.

In October of 1955, a respiratory illness characterized by coughing, sneezing and nasal discharge (coryza), occurred in a colony of chimpanzees being housed at the Walter Reed Army Institute of Research, Forrest Glen Annex in Silver Springs, Maryland.

The facility was constructed in 1953, as a vaccine-production facility, specifically providing living cells from the chimps to grow viruses for experimental vaccine production.

The culprit causing the illness was identified by Morris and colleagues as a virus, and given the name Chimpanzee Coryza Agent (CCA).

Curious about the contagiousness of the virus, the researchers purposefully exposed a second group of adult chimps housed in a nearby facility to the virus. They were able to recreate the respiratory infection, and document the contagiousness of the virus among chimpanzees.

In February of 1956, Morris also reported that a laboratory employee who was working with the chimpanzees who had been purposefully exposed to the virus for research purposes developed a similar “upper respiratory infection” consisting of several days of a runny nose, cough, low-grade fever and headache.

Further investigations documented that the worker (patient B-1) initially tested negative for antibodies to CCA, but positive 14 days later, which suggested the virus was the cause of his illness.

Subsequently, human sera were obtained from patients who presented to Walter Reed Army Medical Center with a variety of illnesses. Antibodies to CCA were unusual in infants and children, but were detected in a group of young adults with respiratory illnesses.

Eight of the young adult patients at the medical center who tested positive for CCA antibodies were barrack mates with patient B-1 and had presumably been exposed to the virus, suggesting the contagious nature of the infection in humans.

One year later in 1957, Chanock and colleagues from Johns Hopkins University Department of Pediatrics received funding to “recover new cytopathogenic agents from infants with severe lower respiratory illness (bronchopneumonia, bronchiolitis and laryngotracheobronchitis.” The funding was provided by the National Institutes of Health, the Rockefeller Foundation and the National Foundation for Infantile Paralysis, who were also the primary sponsors of polio vaccine research at the time.

During the course of their research, Chanock’s team identified two similar viruses in infants diagnosed with pneumonia and croup that were “indistinguishable from an agent associated with coryza in chimpanzees (CCA virus).” The viruses identified in both infants “were not related to the adenovirus group nor to other currently known viruses which cause respiratory illness.”